About 32 million people globally have Alzheimers disease.
Researchers continue to look for a way to stop or slow disease progression.
Researchers from the Icahn School of Medicine at Mount Sinai found that changing certain cellular interactions helps clear out beta-amyloid plaques from the brain, considered a sign of Alzheimer’s disease.
Scientists believe these findings may lead to new treatment options for the condition.
One such team of researchers hails from the Icahn School of Medicine at Mount Sinai in New York. In their new study published in the journal Nature NeuroscienceTrusted Source, researchers found that changing certain cellular interactions helps clear out beta-amyloid plaquesTrusted Source from the brain, considered a sign of Alzheimer’s disease.
Medical News Today spoke to the three senior authors of the study about its findings: Roland H. Friedel, PhD, who is an associate professor of neuroscience and neurosurgery at Icahn, Hongyan Zou, MD, PhD, a professor of neurosurgery and neuroscience at this institution, and Bin Zhang, PhD, who is professor of genetics and genomic sciences at Icahn.
“Alzheimer’s disease remains a medical challenge as currently effective treatment is still not available,” the study authors pointed out.
“Amyloid plaque is a pathological hallmark of Alzheimer’s disease and plaque burden positively correlates with disease severity. Amyloid plaques are neurotoxicTrusted Source — finding ways to help clear out plaques will help reduce neurotoxicity and attenuate neuroinflammation,” they told us.
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